A matching genome is fairly easy to find on earth throughout all animal and plant species because we’re all part of the web of life, related to one another, including the E.T. Also, humans would not exist without the direct contribution of reptiles and birds in the genome which came from E.T. Mammals evolved from them. And most of the plants on the planet were here before we were. Ferns are an example. They are ancient. There used to be huge fern forests on Earth.
Scientists know that DNA variance among even potatoes and people is slim. But potatoes aren’t sentient, that we know of, (potato smarts?) but we are. So what is creating the difference between species if not DNA? A: Evolving tRNA (transfer RNA) that is dynamic due to free will and changes daily according to the oracles that I cite on this blog.
When I note which amino acid tRNA molecules are moving that day it’s the first time they are moving that way in the current time. They move the way they do every 260 days but because we are changing due to our own choices, (Yellow Human free will), the tRNA which turns into DNA in the nucleus is never the same. Thus our kids are impossible to fathom for us parents. They incarnate in groups with agreed-upon agendas. That much I see in generational groups and the astrologers see it also.
A person born on White 13 Wind in 2022 is not the same as a person born in 2019 as White 13 Wind. The Oracle takes care of sprocketing the 365-day solar year with the 260-day galactic spin. The birthday of the person in 2019 is different than the one in 2022, thus their interplanetary astrological line-up is different, and their personality is different. That’s 4D. Things do level out and start matching up in your Mayan Oracle themeplex because we’re dealing with the upper densities of existence, past DNA with the Oracle.
Since being identified in people in 2019, SARS-CoV-2 has gone on to infect a wide range of animal species, wild and domestic. Concerns abound that these species jumps could lead to novel mutations and even harmful new variants.
In a new report, researchers from the University of Pennsylvania’s School of Veterinary Medicine and Perelman School of Medicine found that, for at least one example of apparent interspecies transmission, this crossing the species boundary did not cause the virus to gain a significant number of mutations.
Writing in the journal Viruses, the scientists identified a domestic house cat, treated at Penn Vet’s Ryan Hospital, that was infected with the delta variant of SARS-CoV-2 subsequent to exposure from its owner. The full genome sequence of the virus was a close match to viral sequences circulating in people in the Philadelphia region at the time.
“SARS-CoV-2 has a really incredibly wide host range,” says Elizabeth Lennon, senior author on the work, a veterinarian, and assistant professor at Penn Vet. “What this means to me is that, as SARS-CoV-2 continues to be prevalent in the human population, we need to watch what’s happening in other animal species as well.”
The find is the first published example of the delta variant occurring in a domestic cat in the United States. Notably, the cat’s infection was only identified by testing its fecal matter. A nasal swab did not result in a positive test.
“This did highlight the importance of sampling at multiple body sites,” says Lennon. “We wouldn’t have detected this if we had just done a nasal swab.”
Lennon and colleagues have been sampling dogs and cats for SARS-CoV-2 since early in the pandemic. This particular pet cat, an 11-year-old female, was brought to Ryan Hospital in September with gastrointestinal symptoms. It had been exposed to an owner who had COVID-19—though that owner had been isolated from the cat for 11 days prior to its hospitalization, another household member doing the cat care in the interim.
Working through the Penn Center for Research on Coronaviruses and Other Emerging Pathogens and Perelman School of Medicine microbiologist Frederic Bushman’s laboratory, the team obtained a whole-genome sequence of the cat’s virus.
Sequencing revealed the delta variant, more specifically, the AY.3 lineage. The researchers did not have a sample from the infected owner. Comparing the sequence to the database kept by the Bushman laboratory, however, the cat’s virus was nothing out of the ordinary in terms of the sequences of SARS-CoV-2 circulating in the Delaware Valley region at the time.
“When we looked at a random sampling of human sequences from our geographic area, there wasn’t anything dramatically different about our cat’s sample,” Lennon says. “So, our takeaway was that the cat was not infected by a virus that was somehow highly different.”
Not all variants of SARS-CoV-2 have been equally able to infect a wide range of hosts. For example, the original Wuhan strain could not naturally infect mice; later variants gained that ability. Scientists began seeing infections in cats and dogs from the early days of the pandemic, presumably infected through close contact with their owners.
“The main takeaway here is that as different variants of SARS-CoV-2 emerge, they seem to be retaining the ability to infect a wide range of species,” Lennon says.
While this particular case does not raise alarms for the virus acquiring significant numbers of mutations as it moved between species, Lennon and colleagues, including Bushman and Susan Weiss of Penn’s medical school, hope to continue studying other examples to see how SARS-CoV-2 evolves. Penn Vet’s Institute for Infectious and Zoonotic Disease will facilitate this look at human-animal interactions when it comes to pathogen transmission.
“We know that the SARS-CoV-2 is undergoing changes as it passes between to become more and more transmissible over time,” says Lennon. “We saw that with the omicron variant. It’s host-adapting to people. We also want to know, when other animal species get infected, does the virus start to adapt to those species? And for those viruses that may adapt to a different species, do they still infect humans?”